EMF Blocking in addition to EMF Shielding

· 2 min read
EMF Blocking in addition to EMF Shielding

If you reside in an apartment or house, or you just would like to ensure your home is free of electromagnetic fields, there are a number of methods to reduce exposure. One of the easiest is to reduce your use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from reaching your house. Another method to protect your home against EMF radiation would be to use an RF shielding canopy. It is a type made of net which contains EMF shielding. It's used to prevent EMFs from entering a space. Another option is to get your house equipped with an electrical enclosure. They are referred to as Faraday cages.

A number of studies have proven that the non-ionizing RF EMF has antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to result from the deregulation the cancer stem cell.  Learn here  could be the reason for the long-term response seen in some patients with advanced HCC. However, the mechanism behind AM RF EMF's effect in patients suffering from cancer isn't yet clear.

Effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo was studied in mice. The tumours were divided in three different groups. First, the group that was unaffected to RF EMF. Second group members were exposed to RF EMF at frequencies similar to that used in humans. The third group was exposed RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was compared to that of RCF. The results indicated that cancers treated with HCCMF had significant shrinkage. However, the tumors treated with RCF showed no evidence of shrinkage of the tumor.

The mechanism behind tumor-specific AM RF EMF may be due to the fact that tumour cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effects on HCC cells is controlled by CACNA1H the protein which mediates tumour-specific Ca2+ influx.  https://canvas.instructure.com/eportfolios/2026735/Home/EMF_Block_Paint_plus_EMF_Shielding_2  suggest that CACNA1H could have more broader implications for the treatment and diagnosis of different cancers.


The tumours in those in the group that were unaffected to EMF from radiofrequency, and fed a normal mouse diet. The tumors of the HCCMF group were infected with Huh7 cells after they were 5 to 7 weeks old. The tumors were removed after they had a high burden.

The tumours in the three groups also showed distinct growth curves. The HCCMF-treated tumors had a significant reduction in the size of the tumour after 8 weeks. However, the tumours treated with RCF showed no reduction in size. The difference was significant. The tumors treated by RCF were able to show necrosis, which is common when tumors are that are exposed to RCF.  block emf radiation  is that this necrosis is caused by the lack of oxygen in larger cancers.

In summary, the results indicate that AM RF EMF is a powerful source of anticancer activity in vitro and in vivo. Several studies have shown that AM RF EMF produces measurable tumour shrinkage within HCC patients. The possibility is that the AM EMF causes these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting influence on the growth of HCC tumours in living tissue.