Whether you live in an apartment or a house or simply would like to ensure your home is free of EMFs There are a variety of methods to reduce exposure. One of the most effective is to reduce the use of electronic devices. Browse this site can also turn to EMF blocker paint to block EMF radiation from entering your house. Another easy way to protect your home from EMF radiation is to install a shielding canopy for RF. This is a cloth made of net which contains EMF shielding and is used to prevent EMFs from entering rooms. Another option is to get your home fitted with an electrical enclosure. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF produces antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed involve down-regulation of cancer stem cells. This may account for the long-term effects observed in certain patients suffering from advanced HCC. But, the reason for AM RF EMF's effect in patients suffering from cancer isn't clear.
Aspects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were examined in mice. The tumours were divided into three groups. The first group was not exposed to RF EMF. Second group members were exposed to RF EMF at a frequency that is similar to that used in humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumours was assessed against the effect of RCF. The results showed that the tumours treated with HCCMF showed significant shrinkage. However, the tumors treated with RCF did not show any evidence of tumour shrinkage.
https://canvas.instructure.com/eportfolios/2026741/Home/Details_of_Emf_Blocking_Radiation_2 for tumour-specific AM RF EMF might be based on the fact that tumor cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is mediated through CACNA1H, a protein which regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H could have more broader implications for treatment and diagnosis of many cancers.
The tumours of the control group were not exposed to EMF from RF, and fed a normal diet of mice. The tumors in those in the HCCMF group were injected with Huh7 cells at the time they were five-seven weeks old. The tumors were then killed after they had a high burden.
The tumours in the three groups showed distinct growth curves. The HCCMF-treated tumors saw a significant reduction in the size of the tumour after 8 weeks. However, blocking emf treated with RCF didn't show signs of shrinkage. The difference was substantial. The tumors treated with RCF had necrosis, which is common in tumors that have been that are exposed to RCF. There is a possibility that this necrosis was due to the lack of oxygen in larger tumours.
In sum, the results show the fact that AM EMF exhibits anticancer activity in vitro and in the vivo. Several studies have shown that AM RF EMF produces measurable reduction in tumours in HCC patients. It is possible that AM RF EMF causes these effects because of CACNA1H, a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the development of HCC tumors in vivo.