No matter if you live in an apartment or a house or simply want to keep your home free from EMFs There are plenty options to limit your exposure. One of the most effective is to restrict your use of electronic devices. It is also possible to use EMF block paint to stop EMF radiation from entering your home. Another easy way to protect your house against EMF radiation would be to put up a shielding canopy for RF. It is a type of net that contains EMF shielding and is used to prevent EMFs from entering rooms. Another option is to have your home fitted with a conductive enclosure. They are referred to as Faraday cages.
A number of studies have proven that the non-ionizing RF EMF can cause antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to result from the deregulation of cancer stem cells. This could be the reason for the long-term response seen in certain patients suffering from advanced HCC. However, the mechanism of AM EMF's impact on cancer patients is not yet clear.
Aspects of AM RF EMFs on HCC tumor growth in vivo were studied in mice. The tumors were split in three different groups. First, the group that was unaffected to RF EMF. The second group was exposed to RF EMF at the same frequency to the frequency used by humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on the tumours was evaluated against that of RCF. The results revealed that tumors treated by HCCMF were significantly shrinking. However, the tumours treated with RCF showed no evidence of shrinkage of the tumor.
The mechanism of tumor-specific AM RF EMF could be based on the fact that tumour cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is controlled through CACNA1H which is a protein which mediates tumour-specific Ca2+ influx. Additional hints indicate that CACNA1H may have broader implications for the treatment and diagnosis of different cancers.
The tumours in the control group were not exposed EMF from RF, and fed a normal diet of mice. The tumors in those in the HCCMF group were treated with Huh7 cells after they were between five and seven weeks old. The tumors were removed in cases of excessive burden.
block emf in the three groups also showed different growth curves. The tumors treated with HCCMF showed a significant decrease in size of the tumor after 8 weeks. However, the tumors treated with RCF didn't show reduction in size. The difference was highly significant. The tumors treated with RCF had necrosis that is common in tumours exposed to RCF. It is possible that the necrosis was caused by a lack of oxygen in the larger tumors.
In sum, the results suggest an AM-RF EMF exhibits anticancer activity in vitro and in the vivo. block emf have demonstrated the fact that AM RF EMF produces measurable tumour shrinkage in HCC patients. There is a possibility that AM RF EMF triggers these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained impact on the growth of HCC tumors in vivo.