No matter if you live in an apartment or house or just want to keep your home free of electromagnetic fields There are plenty options to limit your exposure. One of the most effective is to restrict your use of electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from entering your home. Another easy way to protect your house from EMF radiation is to use an RF shielding canopy. It is a type made of net that has EMF shielding. It's used to prevent EMFs from entering a space. Another option is to have your home equipped with an enclosure that is conductive. These devices are called Faraday cages.
Several studies have shown how the non-ionizing energy of RF has antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed be based on the downregulation of cancer stem cells. This could explain the long-term effects observed in certain patients suffering from advanced HCC. However, the mechanism of AM EMF's impact on patients with cancer is not clear.
https://ploughcobweb5.bravejournal.net/post/2023/04/13/Info-about-Emf-Blocking-Radiation on the effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. The tumors were split in three different groups. One group did not have exposure to RF EMF. https://zenwriting.net/findstamp5/emf-block-paint-and-even-emf-shielding was exposed to RF EMF at frequencies similar to the frequency used by humans. The third group was exposed the RF EMF in HCC-specific frequencies. The effects of HCCMF on tumours was assessed against the effect of RCF. The results indicated that tumours treated with HCCMF were significantly shrinking. However, the tumors treated with RCF showed no evidence of tumour shrinkage.
The mechanism of tumour-specific AM RF EMF may be due to the fact that cancer cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is mediated by CACNA1H which is a protein that mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H could have wider implications for treatment and diagnosis of different cancers.
The tumours of the controls were never exposed to RF EMF, and were fed a standard mouse diet. The tumors in those in the HCCMF group were injected with Huh7 cells when they were five-seven weeks old. The tumours were then euthanized in cases of excessive burden.
The tumours in the three groups also showed different growth curves. The tumours treated with HCCMF showed a significant decrease in size of the tumor after eight weeks. However, tumors treated with RCF showed no signs of shrinkage. The difference was substantial. The tumors treated by RCF had necrosis that is common in tumours exposed to RCF. There is a possibility that this necrosis was caused by the lack of oxygen in the more invasive cancers.
In sum, the results show that AM RF EMF exhibits anticancer effects in vitro and in vivo. A number of studies have proven the fact that AM RF EMF produces measurable shrinkage of tumors for HCC patients. The possibility is that the AM EMF produces these effects due to CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the development of HCC tumours in vivo.